|Application ||WB, IHC-P, E, IP|
|Calculated MW||42778 Da|
|Purification||Antisera to human Cdk9 were raised by repeated immunisations of rabbits with highly purified peptide antigen. Whole serum is supplied delipidated.|
|Immunogen||Multiple synthetic peptides corresponding to the C-terminal and N-terminal domains of human CDK9|
|Shelf Life||18 months from date of despatch.|
|Other Names||Cyclin-dependent kinase 9, 220.127.116.11, 18.104.22.168, C-2K, Cell division cycle 2-like protein kinase 4, Cell division protein kinase 9, Serine/threonine-protein kinase PITALRE, Tat-associated kinase complex catalytic subunit, CDK9, CDC2L4, TAK|
|Target/Specificity||Rabbit anti-Human Cdk9 antibody recognizes a ~43 kDa member of the cyclin-dependent protein kinase (CDK) family, CDK9, previously named PITALRE. CDK9 is located primarily in the nucleus but unlike most cdc2-like kinases it does not appear to be cell cycle-regulated. Rabbit anti-Human Cdk9 antibody does not cross react with other cyclin dependent kinases.|
|Preservative & Stabilisers||0.01% Sodium Azide (NaN3)|
|Storage||Store at +4℃ or at -20 ℃.|
|Precautions||Anti-Cdk9 Antibody (Serum) is for research use only and not for use in diagnostic or therapeutic procedures.|
Thousands of laboratories across the world have published research that depended on the performance of antibodies from Abgent to advance their research. Check out links to articles that cite our products in major peer-reviewed journals, organized by research category.
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Provided below are standard protocols that you may find useful for product applications.
1. Graña X et al. (1994) PITALRE, a nuclear CDC2-related protein kinase that phosphorylates the retinoblastoma protein in vitro.Proc Natl Acad Sci U S A. 91 (9): 3834-8. 2. Mayol, X. et al. (1993) Cloning of a new member of the retinoblastoma gene family (pRb2) which binds to the E1A transforming domain.Oncogene. 8 (9): 2561-6. 3. de Falco, G. & Giordano, A. (1998) CDK9 (PITALRE): a multifunctional cdc2-related kinase.J Cell Physiol. 177 (4): 501-6.
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