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>   home   >   Products   >   Primary Antibodies   >   Cell Biology   >   APOBEC3D Antibody (N-term)   

APOBEC3D Antibody (N-term)

Peptide Affinity Purified Rabbit Polyclonal Antibody (Pab)

  • WB - APOBEC3D Antibody (N-term) AP18763a
    APOBEC3D Antibody (N-term)(Cat. #AP18763a) western blot analysis in human placenta tissue lysates (35ug/lane).This demonstrates the APOBEC3D antibody detected the APOBEC3D protein (arrow).
Product Information
  • Applications Legend:
  • WB=Western Blot
  • IHC=Immunohistochemistry
  • IHC-P=Immunohistochemistry (Paraffin-embedded Sections)
  • IHC-F=Immunohistochemistry (Frozen Sections)
  • IF=Immunofluorescence
  • FC=Flow Cytopmetry
  • IC=Immunochemistry
  • ICC=Immunocytochemistry
  • IP=Immunoprecipitation
  • DB=Dot Blot
  • CHIP=Chromatin Immunoprecipitation
  • FA=Fluorescence Assay
  • IEM=Immunoelectronmicroscopy
  • EIA=Enzyme Immunoassay
Primary Accession Q96AK3
Other Accession NP_689639.2
Reactivity Human
Host Rabbit
Clonality Polyclonal
Isotype Rabbit Ig
Calculated MW 46598 Da
Antigen Region 46-72 aa
Additional Information
Other Names DNA dC->dU-editing enzyme APOBEC-3D, A3D, 354-, APOBEC3D
Target/Specificity This APOBEC3D antibody is generated from rabbits immunized with a KLH conjugated synthetic peptide between 46-72 amino acids from the N-terminal region of human APOBEC3D.
Dilution WB~~1:1000
Format Purified polyclonal antibody supplied in PBS with 0.09% (W/V) sodium azide. This antibody is purified through a protein A column, followed by peptide affinity purification.
StorageMaintain refrigerated at 2-8°C for up to 2 weeks. For long term storage store at -20°C in small aliquots to prevent freeze-thaw cycles.
PrecautionsAPOBEC3D Antibody (N-term) is for research use only and not for use in diagnostic or therapeutic procedures.
Protein Information
Function DNA deaminase (cytidine deaminase) which acts as an inhibitor of retrovirus replication and retrotransposon mobility via deaminase-dependent and -independent mechanisms. Exhibits antiviral activity against vif-deficient HIV-1. After the penetration of retroviral nucleocapsids into target cells of infection and the initiation of reverse transcription, it can induce the conversion of cytosine to uracil in the minus-sense single-strand viral DNA, leading to G-to-A hypermutations in the subsequent plus-strand viral DNA. The resultant detrimental levels of mutations in the proviral genome, along with a deamination- independent mechanism that works prior to the proviral integration, together exert efficient antiretroviral effects in infected target cells. Selectively targets single-stranded DNA and does not deaminate double-stranded DNA or single-or double- stranded RNA. May inhibit the mobility of LTR and non-LTR retrotransposons.
Cellular Location Cytoplasm. Cytoplasm, P-body.
Tissue Location Expressed in lymphoid organs. Also detected in non-lymphoid tissues including lung
Research Areas
Citations (0)

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Unknown. Unable to reduce HIV-1 infectivity in vitro.


Zhang, W., et al. PLoS ONE 3 (12), E3963 (2008) :
Dang, Y., et al. J. Virol. 80(21):10522-10533(2006)
Stenglein, M.D., et al. J. Biol. Chem. 281(25):16837-16841(2006)
Conticello, S.G., et al. Mol. Biol. Evol. 22(2):367-377(2005)
Harris, R.S., et al. Nat. Rev. Immunol. 4(11):868-877(2004)

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$ 295.00
$ 99.00
Cat# AP18763a
(40 western blots)
Availability: In Stock
Bulk Size
Seasonal Special on Bulk Order
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