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>   home   >   Products   >   Primary Antibodies   >   Antibody Collections   >   Infectious Disease and Small Molecules   >   Goat Antibody to Respiratory Syncytial Virus (RSV) Horseradish Peroxidase Conjugated   

Goat Antibody to Respiratory Syncytial Virus (RSV) Horseradish Peroxidase Conjugated

Product Information
  • Applications Legend:
  • WB=Western Blot
  • IHC=Immunohistochemistry
  • IHC-P=Immunohistochemistry (Paraffin-embedded Sections)
  • IHC-F=Immunohistochemistry (Frozen Sections)
  • IF=Immunofluorescence
  • FC=Flow Cytopmetry
  • IC=Immunochemistry
  • ICC=Immunocytochemistry
  • IP=Immunoprecipitation
  • DB=Dot Blot
  • CHIP=Chromatin Immunoprecipitation
  • FA=Fluorescence Assay
  • IEM=Immunoelectronmicroscopy
  • EIA=Enzyme Immunoassay
Host Goat
Clonality Polyclonal
Immunogen Human RSV Isolate, Confirmed
Purification IgG fraction covalently coupled to a highly purified preparation of Horseradish Peroxidase (RZ >3). Care is taken to ensure adequate conjugation while preserving maximum enzyme activity. Free enzyme is removed.
Additional Information
Target/Specificity All RSV viral antigens. Reacts well with bovine isolates. Does not react with Para 1-3, Influenza A & B or Adenovirus by IFA. Negative against HEp-2 cells and WI-38 cells.
Preservative 0.002% Thimerosal
Format HRP, Liquid
Storage Short-term (up to 6 months) store at 2-8°C. Long term, aliquot and store at -20°C. Avoid multiple freeze/thaw cycles.
PrecautionsGoat Antibody to Respiratory Syncytial Virus (RSV) Horseradish Peroxidase Conjugated is for research use only and not for use in diagnostic or therapeutic procedures.
Research Areas
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The references listed below are for research purposes only:1.    McLellan, J.S., et al., (2010), “Structure of a more antigenic site on the Respiratory Syncytial Virus fusion glycoprotein in complex with neutralizing antibody 101F, Journal of Virology, 84(23): 12236-12244.2.    Bitko, V., et al., (2007), “Nonstructural Proteins of Respiratory Syncytial Virus Suppress Premature Apoptosis by an NF-kB-Dependent, Interferon-Independent Mechanism and Facilitate Virus Growth”, Journal of Virology, 81(4): 1786-1795.3.    Weltzin, R., et al., (1994), “Intranasal Monoclonal Immunoglobulin A against Respiratory Syncytial Virus Protects against Upper and Lower Respiratory Tract Infections in Mice”, Antimicrobial Agents and Chemotherapy, 38(12): 2785-2791.4.    Ramaswamy, M., et al., (2004), “Specific Inhibition of Type I Interferon Signal Transduction by Respiratory Syncytial Virus”, Am. J. Respir. Cell Mol. Biol., 30: 893-900.5.    Gitiban, N., et al., (2005), “Chinchilla and Murine Models of Upper Respiratory Tract Infections with Respiratory Syncytial Virus”, Journal of Virology, 79(10): 6035-6042.6.    Wright, P.F., et al., (2005), “Growth of Respiratory Syncytial Virus in Primary Epithelial Cells from the Human Respiratory Tract”, Journal of Virology, 79(13): 8651-8654.7.    Monick, MM., et al., (2001), “Respiratory Syncytial Virus Infection Results in Activation of Multiple Protein Kinase C Isoforms Leading to Activation of Mitogen-Activated Protein Kinase”, The Journal of Immunology, 166: 2681-2687.8.    Monick, MM., et al., (2005), “Activation of the Epidermal Growth Factor Receptor by Respiratory Syncytial Virus Results in Increased Inflammation and Delayed Apoptosis”, The Journal of Biological Chemistry”, 280(3): 2147-2158.

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Cat# AR10218
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