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MIPEP Antibody (Center) Blocking PeptideSynthetic peptide

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United States
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Ordering Information
Catalog # Size Availability Price  
BP1459c 0.1 mg 400 ul In Stock $ 45.00 Add to cart
  • Specification
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MIPEP Antibody (Center) Blocking Peptide - Product info

Primary AccessionQ99797
Clone Names70919186
Calculated MW80641 Da

MIPEP Antibody (Center) Blocking Peptide - Additional info

Gene ID 4285
Target/Specificity
The synthetic peptide sequence used to generate the antibody AP1459c was selected from the Center region of human MIPEP. A 10 to 100 fold molar excess to antibody is recommended. Precise conditions should be optimized for a particular assay.
Format
Synthetic peptide was lyophilized with 100% acetonitrile and is supplied as a powder. Reconstitute with 0.1 ml DI water for a final concentration of 1 mg/ml.
Storage
Maintain refrigerated at 2-8°C for up to 6 months. For long term storage store at -20°C.
Precautions
This product is for research use only. Not for use in diagnostic or therapeutic procedures.

MIPEP Antibody (Center) Blocking Peptide - Protein Information

Name MIPEP
Synonyms MIP
Function
Cleaves proteins, imported into the mitochondrion, to their mature size
Cellular Location
Mitochondrion matrix.

MIPEP Antibody (Center) Blocking Peptide - Related products

AM1857a: MIPEP Antibody (Ascites)

AM1857b: MIPEP Antibody

AP1459a: MIPEP Antibody (N-term)

AP1459c: MIPEP Antibody (Center)

AP1459d: MIPEP Antibody

RI13312: MIPEP predesign siRNA

LY11590a: MIPEP Over-expression Lysate

BP1459a: MIPEP Antibody (N-term) Blocking Peptide

BP1459c: MIPEP Antibody (Center) Blocking Peptide

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Provided below are standard protocols that you may find useful for product applications.

BACKGROUND

MIPEP performs the final step in processing a specific class of nuclear-encoded proteins targeted to the mitochondrial matrix or inner membrane. This protein is primarily involved in the maturation of oxidative phosphorylation(OXPHOS)-related proteins. It may contribute to the functional effects of frataxin deficiency and the clinical manifestations of Friedreich ataxia.

REFERENCES

Chew A., Genomics 40:493-496(1997).