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MIPEP Antibody (Center) Blocking PeptideSynthetic peptide
| Country | United States
Ordering Information
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|---|---|---|---|---|
| Catalog # | Size | Availability | Price | |
| BP1459c | 0.1 mg 400 ul | In Stock | $ 45.00 | DISCONTINED INQUIRE CLICK INQUIRE Add to cart |
- Specification
- Citiations : 0
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MIPEP Antibody (Center) Blocking Peptide - Product info | |
| Primary Accession | Q99797 |
| Clone Names | 70919186 |
| Calculated MW | 80641 Da |
MIPEP Antibody (Center) Blocking Peptide - Additional info | |
| Gene ID 4285 | |
| Target/Specificity The synthetic peptide sequence used to generate the antibody AP1459c was selected from the Center region of human MIPEP. A 10 to 100 fold molar excess to antibody is recommended. Precise conditions should be optimized for a particular assay. | |
| Format Synthetic peptide was lyophilized with 100% acetonitrile and is supplied as a powder. Reconstitute with 0.1 ml DI water for a final concentration of 1 mg/ml. | |
| Storage Maintain refrigerated at 2-8°C for up to 6 months. For long term storage store at -20°C. | |
| Precautions This product is for research use only. Not for use in diagnostic or therapeutic procedures. | |
MIPEP Antibody (Center) Blocking Peptide - Protein Information | |
| Name MIPEP | |
| Synonyms MIP | |
| Function Cleaves proteins, imported into the mitochondrion, to their mature size | |
| Cellular Location Mitochondrion matrix. | |
MIPEP Antibody (Center) Blocking Peptide - Related products
AM1857a: MIPEP Antibody (Ascites)
AP1459a: MIPEP Antibody (N-term)
AP1459c: MIPEP Antibody (Center)
RI13312: MIPEP predesign siRNA
LY11590a: MIPEP Over-expression Lysate
MIPEP Antibody (Center) Blocking Peptide - Research Areas
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Provided below are standard protocols that you may find useful for product applications.
BACKGROUND
MIPEP performs the final step in processing a specific class of nuclear-encoded proteins targeted to the mitochondrial matrix or inner membrane. This protein is primarily involved in the maturation of oxidative phosphorylation(OXPHOS)-related proteins. It may contribute to the functional effects of frataxin deficiency and the clinical manifestations of Friedreich ataxia.
REFERENCES
Chew A., Genomics 40:493-496(1997).