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ABAD Antibody (C-term) Blocking PeptideSynthetic peptide
| Country | United States
Ordering Information
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|---|---|---|---|---|
| Catalog # | Size | Availability | Price | |
| BP6307b | 0.1 mg 400 ul | In Stock | $ 45.00 | DISCONTINED INQUIRE CLICK INQUIRE Add to cart |
- Specification
- Citiations : 0
- Reviews
- Protocols
- Backgrounds
ABAD Antibody (C-term) Blocking Peptide - Product info | |
| Primary Accession | Q6IBS9 |
| Clone Names | 4050303 |
ABAD Antibody (C-term) Blocking Peptide - Additional info | |
| Target/Specificity The synthetic peptide sequence used to generate the antibody AP6307b was selected from the C-term region of human ABAD. A 10 to 100 fold molar excess to antibody is recommended. Precise conditions should be optimized for a particular assay. | |
| Format The synthetic peptide was lyophilized with 100% acetonitrile and is supplied as a powder. Reconstitute with 0.1 ml deionized water for a final concentration of 1 mg/ml. | |
| Storage Maintain refrigerated at 2-8°C for up to 6 months. For long term storage store at -20°C. | |
| Precautions This product is for research use only. Not for use in diagnostic or therapeutic procedures. | |
ABAD Antibody (C-term) Blocking Peptide - Protein Information | |
ABAD Antibody (C-term) Blocking Peptide - Related products
AP6307a: ABAD Antibody (Center)
AP6307b: ABAD Antibody (C-term)
AP6307c: HADH2 Antibody (C-term P222)
AP6307d: HADH2 Antibody (Center)
RI12577: HSD17B10 predesign siRNA
LY10129a: HADH2 Over-expression Lysate
BP6307a: ABAD Antibody (Center) Blocking Peptide
BP6307b: ABAD Antibody (C-term) Blocking Peptide
BP6307c: HADH2 Antibody (C-term P222) Blocking Peptide
ABAD Antibody (C-term) Blocking Peptide - Research Areas
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BACKGROUND
Amyloid b-peptide-binding alcohol dehydrogenase (ABAD) is a member of the family of short chain dehydrogenase/reductases; unique among this family, it binds amyloid b-peptide and exhibits enzymatic activity toward a wide variety of substrates including linear alcohols. In an amyloid beta-abundant environment, ABAD appears to trigger cell stress induced by the amyloid peptide.
REFERENCES
FASEB J. 19 (6), 597-598 (2005)J. Mol. Biol. 342 (3), 943-952 (2004)Science 304 (5669), 448-452 (2004)FEBS Lett. 451 (3), 238-242 (1999)J. Biol. Chem. 274 (21), 15014-15019 (1999)