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Anti-IL-2R alpha (Daclizumab), Human IgG1 Antibody
- SPECIFICATION
- CITATIONS
- PROTOCOLS
- BACKGROUND
Application 
| FC |
|---|---|
| Primary Accession | P01589 |
| Reactivity | Human |
| Host | Recombinant |
| Clonality | Monoclonal |
| Isotype | Human IgG1, kappa |
| Calculated MW | 30819 Da |
| Gene ID | 3559 |
|---|---|
| Alias Symbol | IL2RA |
| Other Names | Interleukin-2 receptor subunit alpha; IL-2 receptor subunit alpha; IL-2R subunit alpha; IL-2-RA; IL2-RA; TAC antigen; p55; CD25, Interleukin-2 antibody, IL-2 antibody, IL2 monoclonal antibody, anti-IL2 monoclonal, IL 2 antibody, Interleukin 2 antibody, |
| Appearance | Colorless liquid |
| Formulation | 200 µg affinity purified human antibody in phosphate-buffered saline (PBS) containing 0.02% Proclin 300 |
| Reconstitution & Storage | -20 °C |
| Background Descriptions | |
| Precautions | Anti-IL-2R alpha (Daclizumab), Human IgG1 Antibody is for research use only and not for use in diagnostic or therapeutic procedures. |
| Name | IL2RA |
|---|---|
| Function | Receptor for interleukin-2. The receptor is involved in the regulation of immune tolerance by controlling regulatory T cells (TREGs) activity. TREGs suppress the activation and expansion of autoreactive T-cells. |
| Cellular Location | Membrane; Single-pass type I membrane protein. |
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Provided below are standard protocols that you may find useful for product applications.
Background
Receptor for interleukin-2. The receptor is involved in the regulation of immune tolerance by controlling regulatory T cells (TREGs) activity. TREGs suppress the activation and expansion of autoreactive T-cells.
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